Signal Transduction Pathways, Part A

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applications
approaches
Category=MKFC
chapters
decades
discuss
eq_isMigrated=1
eq_nobargain
expression
field
forward new
gene
hypoxia
insights
luxembourg
meetings
moving
oligonucleotides
part
rapidly
series
therapeutic
transduction
volume
volumes

Product details

  • ISBN 9781573316453
  • Weight: 798g
  • Dimensions: 152 x 230mm
  • Publication Date: 09 Mar 2007
  • Publisher: John Wiley and Sons Ltd
  • Publication City/Country: GB
  • Product Form: Paperback
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Keynote speakers at the cell signaling meeting in Luxembourg have provided chapters on hypoxia signal transduction, phosphoserine/threonine-binding domains, targeting of polycomb repressive complexes, conserved signaling mechanisms in innate immunity, and signal transduction by stress-activated MAP kinases. Other topics included among these reports on recent research are receptor signaling, protein kinase cascades as therapeutic targets, cell death in cancer, inflammation-specific signaling, cell signaling pathways leading to regulated chromatin modifications, and transcriptional control.

The chapters have been published in four volumes (Part A to D), offering a comprehensive overview about this exciting topic.

This first volume focuses on basic research, and the chapters are divided into the following sections: apoptotic cell signaling mechanisms, extracellular matrix interactions, and MAP kinases.

NOTE: Annals volumes are available for sale as individual books or as a journal. For information on institutional journal subscriptions, please visit www.blackwellpublishing.com/nyas.

ACADEMY MEMBERS: Please contact the New York Academy of Sciences directly to place your order (www.nyas.org). Members of the New York Academy of Science receive full-text access to the Annals online and discounts on print volumes. Please visit http://www.nyas.org/MemberCenter/Join.aspx for more information about becoming a member

Marc Diederich is the editor of Signal Transduction Pathways, Part A: Apoptotic and Extracellular Signaling, Volume 1090, published by Wiley.