Type-B Cytochromes: Sensors and Switches
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A01=J.L. Kiel
Assimilatory Nitrate Reductases
Author_J.L. Kiel
Category=PSF
cellular redox signaling
Constitutive No
CTLL-2 Cells
cyclase
Cyclic Adenosine Diphosphate Ribose
Dissimilatory Nitrate Reductase
Disulfide Isomerase Activity
Disulfide Reductase
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guanylate
heme
Hexose Monophosphate Shunt
Johnathan L. Kiel
LPS Treatment
metabolic regulation
nadph
NADPH Diaphorase
NADPH Oxidase
NADPH Oxidase Activity
NADPH oxidase enzymes
nitrate
Nitrate Reductase
Nitric Oxide Reductase
nitric oxide synthase function
nitrite
Nitrite Reductases
NMDA Receptor
Nonheme Iron Sulfur Proteins
oxidase
Oxide Reductase
oxygenase
programmed cell death mechanisms
Protein Kinase
Raw Cell
Redox Chain
redox sensor evolution in biology
reductase
soluble
Soluble Guanylate Cyclase
stress response pathways
System ASC
Thiol Production
Transition Metal
Product details
- ISBN 9781315898315
- Weight: 590g
- Dimensions: 152 x 229mm
- Publication Date: 13 Dec 2017
- Publisher: Taylor & Francis Ltd
- Publication City/Country: GB
- Product Form: Hardback
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This book describes cellular level sensors that act as switches, turning on gene expression and other metabolic processes necessary for cell survival and differentiation. These responses can also initiate programmed cell death or activate latent human immunodeficiency virus or animal leukemia viruses. These redox sensors are nonspecific in sensitivity but specific in response. Unlike ligand/antiligand-type specific sensors, they respond to ionizing and ultraviolet radiations, pH gradients, heat, light, electric and magnetic fields, redox chemicals, mechanical stress, and other nonspecific stressors. The sensors are type-b cytochromes, including NADPH oxidases, NO synthases, and nitrogen oxide reductases. The intense radiation of early pre-biotic earth may have been the evolutionary driving force for the development of their common ancestor.
